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Hur Statiner verkar, och dödar dig celler för cell!

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http://www.amazon.co.uk/Statin-Drugs-Really-Lower-Cholesterol/dp/0615618170

 

How statin drugs really lower cholesterol & kill you one cell at a time

 

"Cholesterol is utterly life vital. We die instantly without it. We need it for every single cell of the body, the muscles, the brain, hormones, bile production, fat digestion, reproduction – it simply cannot be emphasised enough how vital cholesterol is.

It is so vital that the body makes it – the body cannot afford to leave it to chance that we would need to get cholesterol from our diet. This makes cholesterol even more vital to the body than essential fats and protein – as we need to eat these.

Statins stop the body from making the cholesterol that it was designed to make (not entirely, or they would have an immediate 100% death rate).

Statins block something called “the mevalonate pathway”. This is catastrophic. Blocking the mevalonate pathway means that cells cannot replicate or repair themselves properly. Blocking the mevalonate pathway means that every cell in the body dies. The only thing that varies is how long each cell takes to die – some take more time than others.

Nothing can compensate for blocking the mevalonate pathway. Nothing except adding mevalonate back in to the body and we don’t do this. (We don’t even know if we can do this in humans. We shouldn’t block this pathway in the first place.) Adding cholesterol makes no difference, adding CoQ10 makes no difference. Blocking the mevalonate pathway is so flipping serious that anyone who does it should be shot. (I really think using the ‘F’ word here is quite appropriate).

The body tries to respond to the crisis that it detects. As cells realise that their ability to make cholesterol has been impaired, they try to take the cholesterol they need from the blood stream. This lowers blood cholesterol levels and ignorant doctors are happy. They know not what they have done.

There is a second way in which the body tries to save itself – it tries to increase the production of reductase, hoping that this may unblock the mevalonate pathway. It can’t. Hence reductase is both stimulated and inhibited at the same time. Who knows how the body responds to this mechanism being totally confused.

LDL receptor activity and reductase activity increase in parallel. The LDL receptors (the ‘doors’ on each cell responsible for letting LDL in to the cell, with the cholesterol and other good stuff that it is carrying) work harder to try to get LDL from the blood stream into the cells. The reductase production increases to try to reboot the mevalonate pathway, so that cells can make cholesterol as they are designed to do.

The inventors of statins knew what statins were really doing throughout the development of this drug (mevastatin literally means to stop mevalonate – they knew exactly what mevastatin would do). They knew that statins blocked the mevalonate pathway. They knew that this caused cell death. They knew that nothing could compensate for this other than putting the vital mevalonate back. They knew that CoQ10 was affected and this was serious. They patented adding CoQ10 to their statins but then never bothered to add it.

Statins were only originally intended for the 1 in 500 people with Familial Hypercholesterolemia. This would not have enabled drug companies to reach the profit potential of their dreams. Hence cholesterol has been demonised and targets have continually been reset by conflicted bodies so that the norm is no longer the norm and everyone can be told that they need statins.

Ironically, the most serious form of Familial Hypercholesterolemia would receive no benefit from statins anyway. As the extreme form is characterised by LDL receptors working barely at all, even the body going into crisis mode, and trying to take LDL from the blood stream with increased LDL receptor activity, will not work if the LDL receptors are not working sufficiently. Hence the LDL will stay in the blood stream with an extreme sufferer of FH and yet the statin has reduced what little chance the FH sufferer’s body had of making cholesterol within the cell."

http://www.zoeharcombe.com/2013/10/how-statin-drugs-really-lower-cholesterol-and-kill-you-one-cell-at-a-time/

 

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*gnuggar spåkulan*

Statinerna kommer bli vår tids neurosedynskandal.

:angry:

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Ny undersökning/artikel som kopplat högt totalkolesterol till minskad risk för demens

 

Methods: Neuropsychiatric, anthropometric, laboratory, and other assessments were conducted for 392 participants of a 1901 to 1902 birth cohort first examined at age 70. Follow-up examinations were at ages 75, 79, 81, 83, 85, and 88. Information on those lost to follow-up was collected from case records, hospital linkage system, and death certificates. Cox proportional hazards regression examined lipid levels at ages 70, 75, and 79 and incident dementia between ages 70 and 88.

 

"Conclusions: High cholesterol in late life was associated with decreased dementia risk, which is in contrast to previous studies suggesting high cholesterol in mid-life is a risk factor for later dementia. The conflicting results may be explained by the timing of the cholesterol measurements in relationship to age and the clinical onset of dementia."

 

http://www.neurology.org/content/64/10/1689.short

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