Tim Noakes slår tillbaka: Lågfettkost orsakar hjärtsjukdom!

[Peter B]

Tim Noakes har haft 5 dagar i rätten och presenterat sitt material, 30 timmar och 900 slides!!!

 

http://www.thenoakesfoundation.org/news/blog/the-low-fat-diet-does-not-prevent-arterial-disease-it-causes-it-prof-noakes

[Tor H]

Han skal også legge ut alle studiene, men det kan sikkert ta litt tid

[Inez]

Härligt att han har kämpaglöden i behåll, och inte har knäckts av häxjakten

[Zepp]

Han är en pärla.. och rätt rolig att läsa!

 

Och på det så är han rätt ödmjuk.. grejen att de flesta gamla studier är strunt.. rätt många utav dem iallafall!

 

När man går tillbaka och studerar dessa så stämmer rätt sällan data med slutsatserna!

[Peter B]

Mer....

 

http://www.thenoakesfoundation.org/news/blog/noakes-risk-factors-and-insulin-resistance-part-2?news=blog

[Zepp]

"It’s the fatty liver disease, stupid.

More support for Reaven’s unifying hypothesis of chronic disease has recently come from an unexpected source – from those doctors, hepatologists, who specialize in the study of diseases of the liver.

It has been known for some time that the added risks associated with obesity depend, in part, on where that extra fat is stored in the body.  Thus fat that accumulates under the skin – subcutaneous fat – is far less unhealthy than is fat that accumulates within and between the organs in the abdomen, so-called visceral obesity.  The hepatologists have now gone one step further to show that the real killer in visceral obesity is the fat that accumulates within the liver causing NAFLD, a disease that is now also reaching epidemic proportions.

Their work shows that it is NAFLD and not obesity per se that produces the abnormal metabolic state – the atherogenic dyslipidaemia (Tables 1 and 2) – that causes heart disease in those with insulin resistance and the metabolic syndrome.

 

Table 2: The metabolic features of atherogenic dyslipidaemia present in those with NAFLD and insulin resistance

Elevated blood glycated haemoglobin (HbA1c) levels

Elevated fasting blood insulin levels

Elevated fasting blood glucose levels

Hyperinsulinaemia and hyperglycaemia (elevated blood glucose levels) in response to carbohydrate ingestion

Low blood HDL-cholesterol concentrations

High blood triglyceride concentrations

Elevated numbers of small dense LDL-particles

Elevated blood Apolipoprotein B concentrations

 

But the absolutely key point is that dietary carbohydrates and not dietary fat cause NAFLD. For when the insulin resistant eat excess carbohydrates including fructose found in sugar and fruits, they must convert into fat any extra carbohydrate they cannot either use as a fuel or store immediately as carbohydrate in liver or muscles.  Note that all these options are severely reduced in those with insulin resistance.  Instead under the action of insulin – the fat-building hormone –  that fat is stored, initially as fat in the liver.  But as NAFLD develops, insulin resistance worsens, hyperinsulinaemia increases, atherogenic dyslipidaemia deteriorates and the seeds for the chronic diseases of obesity, diabetes, heart disease, NAFLD and perhaps cancer and dementia are sown.

Thus it is that dietary carbohydrates and not dietary fat that is the direct cause of this group of chronic diseases in those with insulin resistance."

[Tor H]

Nå har Tim Noakes lagt ut to videoer, introduksjon og del 1 fra rettsmøtet:

 

http://www.thenoakesfoundation.org/news/blog/tim-noakes-hpcsa-deposition-2016-introduction-and-part-1?news=blog

 

Dette kan bli spennende